A protein ripatrice damage to the heart
The protein thymosin beta-4 activates the stem cells to develop into cardiomyocytes, epicardium and reform so damaged heart tissue after infarction.
Nature has been published to a survey that explains the remedial effects of a protein called thymosin beta-4, on the damage caused infarction.
The thymosin beta-4 seems to be able to activate the stem cells nell’epicardio, the membrane that lines the outer surface of the heart, and they fit in where they differentiate into heart muscle cells that form the heart tissue, cardiomyocytes. In addition, the protein would also be able to reduce the portion of the scar tissue that forms after the infarction.
Currently, the experiment was performed only on mice, but the research director Paul Riley, professor of molecular cardiology at University College London, held to explain: “In mice we found an increase of 25% of the amount of blood that the heart pumps with each heartbeat to the outside of the left ventricle. ”
The authors of the hope that the drug enters the market about ten years to allow patients at risk of cardiac infarction to make repairs much more efficient.
The medical director of the British Heart Foundation, Peter Weisberg says, however, that does not mean that you can achieve the same results of research on human beings, but of course the protein in question will make better quality of life of those heart attacks.